DEPRESSED GLUCOSE CONSUMPTION AT REPERFUSION FOLLOWING BRAIN ISCHEMIA DOES NOT CORRELATE WITH MITOCHONDRIAL DYSFUNCTION AND DEVELOPMENT OF INFARCTION: AN IN VIVO POSITRON EMISSION TOMOGRAPHY STUDY

DEPRESSED GLUCOSE CONSUMPTION AT REPERFUSION FOLLOWING BRAIN ISCHEMIA DOES NOT CORRELATE WITH MITOCHONDRIAL DYSFUNCTION AND DEVELOPMENT OF INFARCTION: AN IN VIVO POSITRON EMISSION TOMOGRAPHY STUDY

.,
2009
References: 
Depressed glucose consumption at reperfusion following brain ischemia does not correlate with mitochondrial dysfunction and development of infarction: an in vivo positron emission tomography study.Martín A, Rojas S, Pareto D, Santalucia T, Millán O, Abaso
  • Associated Personnel

    Anna M Planas Obradors

    Research Professor
  • Associated Research Groups
  • Associated Departments

    Neuroscience and Experimental Therapeutics.

    The department studies the biological bases and molecular mechanisms of brain aging and brain pathologies, including neurodegenerative, cerebrovascular, and neuroinflammatory diseases. In the same way, it addresses the study of brain circuits and synaptic processes involved in neuropsychiatric disorders such as depression and schizophrenia.

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