B-CELL TRANSLOCATION GENE 2 IS OVER-EXPRESSED IN PERI-INFARCT NEURONS AFTER ISCHAEMIC STROKE
CELL THERAPIES FOR INFLAMMATORY BOWEL DISEASES
LIPOPROTEIN LIPASE IS NITRATED IN VIVO AFTER LIPOPOLYSACCHARIDE CHALLENGE
ROLE OF DIFFERENT MONOAMINE RECEPTORS CONTROLLING MK-801-INDUCED RELEASE OF SEROTONIN AND GLUTAMATE IN THE MEDIAL PREFRONTAL CORTEX: RELEVANCE FOR ANTIPSYCHOTIC ACTION
SIRTUIN AND RESVERATROL. CHAPTER 22, PP. 329-340. IN: MICRONUTRIENTS AND BRAIN HEATLH. EDS: PACKER L, SIES H, EGGERSDORFER M, CADENAS E. CRC PRESS, BOCA RATON, FL, USA - CAPITULO LIBRO
ASTROCYTES ARE VERY SENSITIVE TO DEVELOP INNATE IMMUNE RESPONSES TO LIPID-CARRIED SHORT INTERFERING RNA
HARMS AND BENEFITS OF LYMPHOCYTE SUBPOPULATIONS IN PATIENTS WITH ACUTE STROKE
LACK OF CB1 RECEPTOR ACTIVITY IMPAIRS SEROTONERGIC NEGATIVE FEEDBACK
REACTIVE OXYGEN SPECIES MEDIATE LIVER INJURY THROUGH PARENCHYMAL NUCLEAR FACTOR-KAPPA B INACTIVATION IN PROLONGED ISCHEMIA/REPERFUSION
El splicing alternativo de H-Ras en mamíferos une varios factores de splicing a procesos cancerígenos.
Tissue plasminogen activator induces pancreas cancer cell proliferation by a non-catalytic mechanism that requires ERK1/2 activation through Epidermal Growth Factor Receptor and Annexin A2
"UPREGULATION OF CCAAT/ENHANCER BINDING PROTEIN BETA IN ACTIVATED ASTROCYTES AND MICROGLIA."
''DISTRIBUTION OF CLOSTRIDIUM PERFRINGENS EPSILON TOXIN IN THE BRAINS OF ACUTELY INTOXICATED MICE AND ITS EFFECT PON GLIAL CELLS.''
''CCAAT/ENHANCER BINDING PROTEIN ALPHA IS DOWNREGULATED BY TOLL-LIKE RECEPTOR AGONISTS IN MICROGLIAL CELLS.''
Trimethylguanosine nucleoside inhibits cross-linking between Snurportin 1 and m3G-capped U1 snRNP
Tissue plasminogen activator mediates amyloid induced neurotoxicity via sustained Erk1/2 activation.
The tissue plasminogen activator system in murine exocrine pancreas cancer:selective expression in ductal tumors and contribution to cancer progression.
New insights into the tPA-Annexin A2 interaction: is Annexin A2 Cys8 the sole requirement for this association?
Alternative splicing of the human proto-oncogene c-H-ras renders a new Ras family protein which trafficks to cytoplasm and nucleus.
HnRNP A1 and SR proteins regulate the alternative splicing of proto-oncogen c-H-ras, balancing the relative abundance of two alternative H-Ras proteins, p19 and p21.
