''IGL-1 SOLUTION REDUCES ENDOPLASMIC RETICULUM STRESS AND APOPTOSIS IN RAT LIVER TRANSPLANTATION.''
''ISCHEMIC PRECONDITIONING REDUCES ENDOPLASMIC RETICULUM STRESS AND UPREGULATES HYPOXIA INDUCIBLE FACTOR-1? IN ISCHEMIC KIDNEY: THE ROLE OF NITRIC OXIDE.''
"ADDITION OF CARVEDILOL TO UNIVERSITY WISCONSIN SOLUTION IMPROVES RAT STEATOTIC AND NONSTEATOTIC LIVER PRESERVATION."
"MATRIX METALLOPROTEINASE 2 IN REDUCED-SIZE LIVER TRANSPLANTATION: BEYOND THE MATRIX."
"INSULIN LIKE GROWTH FACTOR-1 INCREASES FATTY LIVER PRESERVATION IN IGL-1 SOLUTION."
"INHIBITORY ACTION OF WNT TARGET GENE OSTEOPONTIN ON MITOCHONDRIAL CYTOCHROME C RELEASE DETERMINES RENAL ISCHEMIC RESISTANCE."
"IMPROVED RAT STEATOTIC AND NONSTEATOTIC LIVER PRESERVATION BY THE ADDITION OF EPIDERMAL GROWTH FACTOR AND INSULIN-LIKE GROWTH FACTOR-I TO UNIVERSITY OF WISCONSIN SOLUTION."
"HYPOXIA INDUCIBLE FACTOR-1ALPHA ACCUMULATION IN STEATOTIC LIVER PRESERVATION: ROLE OF NITRIC OXIDE."
"PHARMACOLOGICAL STRATEGIES AGAINST COLD ISCHEMIA REPERFUSION INJURY."
"NEW INSIGHTS INTO FATTY LIVER PRESERVATION USING INSTITUTE GEORGES LOPEZ PRESERVATION SOLUTION."
"SERUM NEUTROPHIL GELATINASE-ASSOCIATED LIPOCALIN IN PATIENTS WITH COLORECTAL LIVER METASTASES: PRELIMINARY RESULTS OF AN EXPLORATORY PROSPECTIVE STUDY."
"ROLE OF OXIDATIVE STRESS AND ADENOSINE NUCLEOTIDES IN THE LIVER OF AGING RATS."
THE REG4 GENE, AMPLIFIED IN THE EARLY STAGES OF PANCREATIC CANCER DEVELOPMENT, IS A PROMISING THERAPEUTIC TARGET
ARE ANGIOTENSIN II RECEPTOR ANTAGONISTS USEFUL STRATEGIES IN STEATOTIC AND NONSTEATOTIC LIVERS IN CONDITIONS OF PARTIAL HEPATECTOMY UNDER ISCHEMIA-REPERFUSION?
EFFECT OF ANGIOTENSIN II AND BRADYKININ INHIBITION IN RAT REDUCED-SIZE LIVER TRANSPLANTATION
CISPLATIN UPREGULATES MITOCHONDRIAL NITRIC OXIDE SYNTHASE AND PEROXYNITRITE FORMATION TO PROMOTE RENAL INJURY
INSULIN-LIKE GROWTH FACTOR AND EPIDERMAL GROWTH FACTOR TREATMENT: NEW APPROACHES TO PROTECTING STEATOTIC LIVERS AGAINST ISCHEMIA-REPERFUSION INJURY.
PREVENTION OF I/R INJURY IN FATTY LIVERS BY ISCHEMIC PRECONDITIONING IS ASSOCIATED WITH INCREASED MITOCHONDRIAL TOLERANCE: THE KEY ROLE OF ATPSYNTHASE AND MITOCHONDRIAL PERMEABILITY TRANSITION.
TUBULAR EPITHELIAL CELLS TRANSFECTED WITH HHGF COUNTERACTS MONOCYTE CHEMOTACTIC PROTEIN-1 UP-REGULATION AFTER HYPOXIA/REOXYGENATION INSULT.
THERAPEUTIC TARGETS IN LIVER TRANSPLANTATION: ANGIOTENSIN II IN NONSTEATOTIC GRAFTS AND ANGIOTENSIN-(1-7) IN STEATOTIC GRAFTS