''ASTROCYTE TLR4 ACTIVATION INDUCES A PROINFLAMMATORY ENVIRONMENT THROUGH THE INTERPLAY BETWEEN MYD88-DEPENDENT NF?B SIGNALING, MAPK, AND JAK1/STAT1 PATHWAYS.''

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Home » Publications » ''ASTROCYTE TLR4 ACTIVATION INDUCES A PROINFLAMMATORY ENVIRONMENT THROUGH THE INTERPLAY BETWEEN MYD88-DEPENDENT NF?B SIGNALING, MAPK, AND JAK1/STAT1 PATHWAYS.''

''ASTROCYTE TLR4 ACTIVATION INDUCES A PROINFLAMMATORY ENVIRONMENT THROUGH THE INTERPLAY BETWEEN MYD88-DEPENDENT NF?B SIGNALING, MAPK, AND JAK1/STAT1 PATHWAYS.''

GORINA R, FONT-NIEVES M, MÁRQUEZ-KISINOUSKY L, SANTALUCIA T, PLANAS AM.,
2011
References: 
Glia. 2011 Feb;59(2):242-55.
  • Associated Personnel

    Anna M Planas Obradors

    Research Professor
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  • Associated Departments

    Neuroscience and Experimental Therapeutics.

    The department studies the biological bases and molecular mechanisms of brain aging and brain pathologies, including neurodegenerative, cerebrovascular, and neuroinflammatory diseases. In the same way, it addresses the study of brain circuits and synaptic processes involved in neuropsychiatric disorders such as depression and schizophrenia.

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