PINDOLOL AUGMENTATION ENHANCES RESPONSE OUTCOMES IN FIRST DEPRESSIVE EPISODES
EFFECT OF ACUTE AND CHRONIC WHOLE-BODY VIBRATION EXERCISE ON SERUM INSULIN-LIKE GROWTH FACTOR-1 LEVELS IN WOMEN WITH FIBROMYALGIA
ESTIMATION OF THE EFFECTIVE ORIENTATION OF THE SHG SOURCE IN PRIMARY CORTICAL NEURONS
NIMODIPINE INHIBITS TMB-8 POTENTIATION OF AMPA-INDUCED HIPPOCAMPAL NEURODEGENERATION
URINE PROTEOMICS TO DETECT BIOMARKERS FOR CHRONIC ALLOGRAFT DYSFUNCTION
DISCOVERY OF LIPOPROTEIN LIPASE PI ISOFORMS AND CONTRIBUTIONS TO THEIR CHARACTERIZATION.
ENHANCED FREE CHOLESTEROL, SREBP-2 AND STAR EXPRESSION IN HUMAN NASH
NEURONAL IN VITRO MODELS FOR THE ESTIMATION OF ACUTE SYSTEMIC TOXICITY
TUBULAR EPITHELIAL CELLS TRANSFECTED WITH HHGF COUNTERACTS MONOCYTE CHEMOTACTIC PROTEIN-1 UP-REGULATION AFTER HYPOXIA/REOXYGENATION INSULT.
El splicing alternativo de H-Ras en mamíferos une varios factores de splicing a procesos cancerígenos.
Tissue plasminogen activator induces pancreas cancer cell proliferation by a non-catalytic mechanism that requires ERK1/2 activation through Epidermal Growth Factor Receptor and Annexin A2
''DISTRIBUTION OF CLOSTRIDIUM PERFRINGENS EPSILON TOXIN IN THE BRAINS OF ACUTELY INTOXICATED MICE AND ITS EFFECT PON GLIAL CELLS.''
''CCAAT/ENHANCER BINDING PROTEIN ALPHA IS DOWNREGULATED BY TOLL-LIKE RECEPTOR AGONISTS IN MICROGLIAL CELLS.''
"UPREGULATION OF CCAAT/ENHANCER BINDING PROTEIN BETA IN ACTIVATED ASTROCYTES AND MICROGLIA."
Trimethylguanosine nucleoside inhibits cross-linking between Snurportin 1 and m3G-capped U1 snRNP
Tissue plasminogen activator mediates amyloid induced neurotoxicity via sustained Erk1/2 activation.
The tissue plasminogen activator system in murine exocrine pancreas cancer:selective expression in ductal tumors and contribution to cancer progression.
New insights into the tPA-Annexin A2 interaction: is Annexin A2 Cys8 the sole requirement for this association?
HnRNP A1 and SR proteins regulate the alternative splicing of proto-oncogen c-H-ras, balancing the relative abundance of two alternative H-Ras proteins, p19 and p21.
Alternative splicing of the human proto-oncogene c-H-ras renders a new Ras family protein which trafficks to cytoplasm and nucleus.
