Human Lysyl Oxidase Over-Expression Enhances Baseline Cardiac Oxidative Stress but Does Not Aggravate ROS Generation or Infarct Size Following Myocardial Ischemia-Reperfusion.
High NOR-1 (Neuron-Derived Orphan Receptor 1) expression strengthens the vascular wall response to angiotensin ii leading to aneurysm formation in mice.
Microsomal Prostaglandin E Synthase-1 (mPGES-1) is involved in the metabolic and cardiovascular alterations associated with obesity
Interferon stimulated gene 15 pathway is a novel mediator of endothelial dysfunction and aneurysms development in angiotensin II infused mice through increased oxidative stress.
NR4A3: A Key Nuclear Receptor in Vascular Biology, Cardiovascular Remodeling, and Beyond
Targeting Tyrosine Hydroxylase for Abdominal Aortic Aneurysm: Impact on Inflammation, Oxidative Stress, and Vascular Remodeling.
Earth: the planet of the annexins La Tierra: el planeta de las anexinas
Oxidative Stress and Inflammatory Markers in Abdominal Aortic Aneurysm
Rolipram prevents the formation of abdominal aortic aneurysm (AAA) in mice: PDE4B as a target in AAA.
Cells in cardiovascular disease: using diversity to confront adversity.
Deletion or Inhibition of NOD1 Favors Plaque Stability and Attenuates Atherothrombosis in Advanced Atherogenesis
Neuron-derived Orphan receptor-1 Modulates Cardiac Gene Expression and Exacerbates Angiotensin II-induced Cardiac Hypertrophy
The role of lysyl oxidase enzymes in cardiac function and remodeling.
The role of mitochondrial oxidative stress in the metabolic alterations in diet induced obesity in rats.
Opposite effects of moderate and extreme Cx43 deficiency in conditional Cx43-deficient mice on angiotensin II-induced cardiac fibrosis.
Emerging roles of lysyl oxidases in the cardiovascular system: new concepts and therapeutic challenges.
Enhanced endoplasmic reticulum and mitochondrial stress in abdominal aortic aneurysm
Endothelial NOD1 directs myeloid cell recruitment in atherosclerosis through VCAM-1.
OxLDL receptor in lymphocytes prevents atherosclerosis and predicts subclinical disease.
The nuclear receptor NOR-1 modulates redox homeostasis in human vascular smooth muscle cells.