The CD200R1 microglial inhibitory receptor as a therapeutic target in the MPTP model of Parkinson's disease
The CD200R1 microglial inhibitory receptor as a therapeutic target in the MPTP model of Parkinson’s disease
Microglial hyperreactivity evolved to immunosuppression in the hippocampus of a mouse model of accelerated aging and Alzheimer's Disease traits.
RNA-Seq transcriptomic profiling of primary murine microglia treated with LPS or LPS + IFNγ
Alterations in CD200-CD200R1 system during EAE already manifest at presymptomatic stages.
Myeloid C/EBPβ deficiency reshapes microglial gene expression and is protective in experimental autoimmune encephalomyelitis
The receptor CMRF35-like molecule (CLM-1) enhances the production of LPS-induced production of pro-inflammatory mediators during microglial activation.
Parkinsonian Neurotoxicants Impair the Anti-Inflammatory Response Induced by IL4 in Glial Cells: Involvement of the CD200-CD200R1 Ligand-Receptor Pair
CCAAT/enhancer Binding Protein δ Is a Transcriptional Repressor of α-synuclein
CD200 Is Up-Regulated in R6/1 Transgenic Mouse Model of Huntington's Disease
Parkinsonian Neurotoxins Impair the Pro-inflammatory Response of Glial Cells
Biosafety assessment of conducting nanostructured materials by using co-cultures of neurons and astrocytes
''CD200R1 AND CD200 EXPRESSION ARE REGULATED BY PPAR-GAMMA; IN ACTIVATED GLIAL CELLS.''
''CCAAT/ENHANCER BINDING PROTEIN DELTA REGULATES PRO-INFLAMMATORY GENE EXPRESSION IN GLIAL ACTIVATION: POTENTIAL ROLE IN AMYOTROPHIC LATERAL SCLEROSIS.''
''CCAAT/ENHANCER BINDING PROTEIN BETA REGULATES PTGES EXPRESSION AND PGE2 PRODUCTION IN ACTIVATED MICROGLIAL CELLS.''
''C/EBP BETA EXPRESSION IN ACTIVATED MICROGLIA IN AMYOTROPHIC LATERAL SCLEROSIS.''
''INHIBITION OF CD200R1 EXPRESSION BY C/EBP BETA IN REACTIVE MICROGLIAL CELLS.''
''PCAF REGULATES THE STABILITY OF THE TRANSCRIPTIONAL REGULATOR AND CYCLIN-DEPENDENT KINASE INHIBITOR P27 KIP1.''
''MODELLING NEUROINFLAMMATION IN VITRO: A TOOL TO TEST THE POTENTIAL NEUROPROTECTIVE EFFECT OF ANTI-INFLAMMATORY AGENTS.''
''TISSUE PLASMINOGEN ACTIVATOR INDUCES MICROGLIAL INFLAMMATION VIA A NON-CATALYTIC MOLECULAR MECHANISM INVOLVING ACTIVATION OF MAPKS AND AKT SIGNALLING PATHWAYS AND ANNEXINA2 AND GALECTIN1 RECEPTORS.''