April 11 - World Parkinson's Disease Day at the IIBB (video)

Parkinson's disease is the second most common neurodegenerative disease after Alzheimer’s disease and is characterized by a progressive loss of neurons involved in the control of movement.

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The IIBB publishes 12 offers for JAE intro 2021 candidates

More info and details are available here.

Applications deadline: 08/04/2021

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Modified oligonucleotide targets mutant alpha-synuclein forms in Parkinson's disease

  • The oligonucleotide, designed to act only on dopaminergic and noradrenergic neurons, reduces the production and accumulation of the mutant alpha-synuclein protein, a key protein in the development of Parkinson's disease

  • The work is led by the Barcelona Biomedical Research Institute of the CSIC and IDIBAPS

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A novel cholesterol-mediated mechanism of regulation of mitophagy in Alzheimer’s disease

The study recently published in Molecular Neurodegeneration journal with Vicente Roca as the first author and directed by

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IIBB researchers contribute to an international guide on the regulation of autophagy

They have participated in the 4th edition of an international consensus on the interpretation, mechanisms and various multidisciplinary aspects of autophagy.

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Dr. Hove-Madsen (IIBB) participates in an international and multidisciplinary project that identifies a molecular mechanism of the cardiac alternans, one of the causes of severe cardiac arrhythmia and sudden death

An international team of multidisciplinary researchers has identified one of the molecular mechanisms underlying cardiac alternans, an alteration of the heart rhythm that facilitates the induction of ventricular fibrillation, a potentially fatal arrhythmia. Furthermore, they show that people with loss of ryanodine receptor (RyR) function are prone to induction of ventricular fibrillation and sudden death caused by cardiac alternans.

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The IIBB joins the initiative #11F International Day of Women and Girls in Science

The importance of including women in all sciences at all levels

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