H-Ras Pre-mRNA Contains A Regulatory Non-coding RNA
Downregulation of p68 RNA Helicase (DDX5) Activates a Survival Pathway Involving mTOR and MDM2 Signals.
The role of p19 and p21 H-Ras proteins and mutants in miRNA expression in cancer and a Costello syndrome cell model.
''THE ROLE OF P19 AND P21 H-RAS PROTEINS AND MUTANTS IN MIRNA EXPRESSION IN CANCER AND A COSTELLO SYNDROME CELL MODEL.''
P68 RNA helicase (DDX5) fine-tunes dynamic localization and activity of cis- and trans-splicing components of the alternative splicing of H-ras.
P19 H-ras induces G1/S phase delay maintaining cells in a reversible quiescence state.
P19 H-RAS INDUCES G1/S PHASE DELAY MAINTAINING CELLS IN A REVERSIBLE QUIESCENCE STATE
El splicing alternativo de H-Ras en mamíferos une varios factores de splicing a procesos cancerígenos.
Trimethylguanosine nucleoside inhibits cross-linking between Snurportin 1 and m3G-capped U1 snRNP
Alternative splicing of the human proto-oncogene c-H-ras renders a new Ras family protein which trafficks to cytoplasm and nucleus.
HnRNP A1 and SR proteins regulate the alternative splicing of proto-oncogen c-H-ras, balancing the relative abundance of two alternative H-Ras proteins, p19 and p21.
Study of the 2719 mutant of the c-H-ras oncogen in a bi-intronic alternative splicing system.
Modulation in vitro of H-ras oncogene expresión by trans-splicing
Post-transcriptional regulation of rat carnitine octanoyltransferase.
Localization of an exonic splicing enhancer responsible for mammalian natural trans-splicing
Alternative splicing of c-H-ras, a system to study different aspects of the splicing mechanism.
Processing of carnitine octanoyltransferase pre-mRNAs by cis and trans-splicing.